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- W1981139440 abstract "The purpose of this present study was to explore the therapeutic potential of prostaglandins E1 and E2 on the systemic inflammatory response evoked by endotoxin. Since interleukin-18, a monocyte-derived cytokine, is increased during sepsis, decreasing the production of interleukin-18 is important in treating this condition. Prostaglandin E1 and E2 inhibited interleukin-18 production in human monocytes treated with lipopolysaccharide and prostanoid IP-, EP2- and EP4-receptor agonists mimicked the effects of prostaglandins E1 and E2. Therefore, prostanoid IP, EP2- and EP4-receptors might be involved in the decrease in interleukin-18 production during sepsis." @default.
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- W1981139440 date "2005-07-01" @default.
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- W1981139440 title "Prostaglandins E1 and E2 inhibit lipopolysaccharide-induced interleukin-18 production in monocytes" @default.
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- W1981139440 doi "https://doi.org/10.1016/j.ejphar.2005.05.020" @default.
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