FRINK Linked Data Fragments server

Matches in SemOpenAlex for { <https://semopenalex.org/work/W1981569598> ?p ?o ?g. }

• W1981569598 endingPage "119" @default.
• W1981569598 startingPage "114" @default.
• W1981569598 abstract "Clinical and experimental data suggest that T helper (TH) cells are involved in the pathogenicity of experimental autoimmune encephalomyelitis (EAE) and multiple sclerosis (MS), but it is unlikely that they are directly responsible for the observed demyelination and axonal loss. Instead, the cell population that targets the destruction of oligodendrocytes and axons, and the mechanism exploited by central nervous system (CNS)-invading encephalitogenic TH cells to instruct these cells to mediate tissue damage, are still under debate. Mature myeloid cells form a prominent component of the neuroinflammatory infiltrates and are the suspected culprits behind the CNS injury due to their arsenal of toxic factors. Here, we describe the process of encephalitogenic TH cell activation followed by their entry into the CNS and discuss how pathogenic TH cells influence the myeloid compartment." @default.
• W1981569598 created "2016-06-24" @default.
• W1981569598 creator A5010172815 @default.
• W1981569598 creator A5025846021 @default.
• W1981569598 creator A5051595544 @default.
• W1981569598 date "2013-03-01" @default.
• W1981569598 modified "2023-10-08" @default.
• W1981569598 title "Communication between pathogenic T cells and myeloid cells in neuroinflammatory disease" @default.
• W1981569598 cites W1584119939 @default.
• W1981569598 cites W1596173029 @default.
• W1981569598 cites W1788325364 @default.
• W1981569598 cites W1973097991 @default.
• W1981569598 cites W1975110303 @default.
• W1981569598 cites W1976591271 @default.
• W1981569598 cites W1979358011 @default.
• W1981569598 cites W1980748156 @default.
• W1981569598 cites W1981688486 @default.
• W1981569598 cites W1982021670 @default.
• W1981569598 cites W1987628377 @default.
• W1981569598 cites W1987931077 @default.
• W1981569598 cites W1991070647 @default.
• W1981569598 cites W1993741037 @default.
• W1981569598 cites W1995644527 @default.
• W1981569598 cites W1997245227 @default.
• W1981569598 cites W1998895238 @default.
• W1981569598 cites W2001235261 @default.
• W1981569598 cites W2002433576 @default.
• W1981569598 cites W2002967638 @default.
• W1981569598 cites W2006249161 @default.
• W1981569598 cites W2009404162 @default.
• W1981569598 cites W2015627239 @default.
• W1981569598 cites W2020756145 @default.
• W1981569598 cites W2026578487 @default.
• W1981569598 cites W2035213168 @default.
• W1981569598 cites W2035939390 @default.
• W1981569598 cites W2040547343 @default.
• W1981569598 cites W2043683714 @default.
• W1981569598 cites W2046413321 @default.
• W1981569598 cites W2050531099 @default.
• W1981569598 cites W2053313675 @default.
• W1981569598 cites W2054235049 @default.
• W1981569598 cites W2054254705 @default.
• W1981569598 cites W2055021280 @default.
• W1981569598 cites W2062762880 @default.
• W1981569598 cites W2063680739 @default.
• W1981569598 cites W2074154043 @default.
• W1981569598 cites W2076889715 @default.
• W1981569598 cites W2081845571 @default.
• W1981569598 cites W2084017668 @default.
• W1981569598 cites W2084388036 @default.
• W1981569598 cites W2089470402 @default.
• W1981569598 cites W2090197666 @default.
• W1981569598 cites W2092743770 @default.
• W1981569598 cites W2092975931 @default.
• W1981569598 cites W2093439715 @default.
• W1981569598 cites W2096618637 @default.
• W1981569598 cites W2103076363 @default.
• W1981569598 cites W2103600875 @default.
• W1981569598 cites W2104777875 @default.
• W1981569598 cites W2106471879 @default.
• W1981569598 cites W2106624302 @default.
• W1981569598 cites W2107235439 @default.
• W1981569598 cites W2108373826 @default.
• W1981569598 cites W2110722277 @default.
• W1981569598 cites W2112880160 @default.
• W1981569598 cites W2115396398 @default.
• W1981569598 cites W2121236896 @default.
• W1981569598 cites W2127671643 @default.
• W1981569598 cites W2131761283 @default.
• W1981569598 cites W2134453100 @default.
• W1981569598 cites W2136158798 @default.
• W1981569598 cites W2141214625 @default.
• W1981569598 cites W2142017607 @default.
• W1981569598 cites W2149703418 @default.
• W1981569598 cites W2150957292 @default.
• W1981569598 cites W2151363901 @default.
• W1981569598 cites W2153274751 @default.
• W1981569598 cites W2156546316 @default.
• W1981569598 cites W2156642970 @default.
• W1981569598 cites W2157837362 @default.
• W1981569598 cites W2162771786 @default.
• W1981569598 cites W2164392039 @default.
• W1981569598 cites W2165598209 @default.
• W1981569598 cites W2166676682 @default.
• W1981569598 cites W2166787544 @default.
• W1981569598 cites W2167379455 @default.
• W1981569598 cites W2171467864 @default.
• W1981569598 cites W2299336915 @default.
• W1981569598 cites W2344751406 @default.
• W1981569598 cites W4229932900 @default.
• W1981569598 doi "https://doi.org/10.1016/j.it.2012.09.007" @default.
• W1981569598 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/23116549" @default.
• W1981569598 hasPublicationYear "2013" @default.
• W1981569598 type Work @default.
• W1981569598 sameAs 1981569598 @default.
• W1981569598 citedByCount "57" @default.
• W1981569598 countsByYear W19815695982013 @default.
• W1981569598 countsByYear W19815695982014 @default.

• next