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- W1981626239 abstract "Emergence of hepatitis B virus (HBV) mutants defective in hepatitis B e antigen (HBeAg) expression is usually a consequence of nonsense or frameshift mutations owing to a single nucleotide change or insertion/deletion in the precore region. In two rare HBe-minus HBV mutants, a nonsense mutation at precore codon 28 and a frameshift mutation at codon 29 were respectively associated with additional nucleotide substitution(s). Were these additional changes accidentally associated with the HBe-abolishing mutations or did they play a role in viral replication? Construction of artificial mutants followed by transfection experiments revealed the importance of these sequence changes for the efficient packaging of pregenomic RNA. These results can be best explained by the presence of a base-paired region of the viral pregenome encapsidation signal overlapping HBe-coding sequences, which can tolerate primary sequence changes." @default.
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- W1981626239 date "1994-01-01" @default.
- W1981626239 modified "2023-09-28" @default.
- W1981626239 title "Base Pairing in the Pregenome Encapsidation Signal of HBV: A Clue for the Prevalence of Naturally Occurring HBeAg-minus Precore Mutations" @default.
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- W1981626239 doi "https://doi.org/10.1007/978-4-431-68255-4_67" @default.
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