Matches in SemOpenAlex for { <https://semopenalex.org/work/W1981804246> ?p ?o ?g. }
- W1981804246 endingPage "15" @default.
- W1981804246 startingPage "1" @default.
- W1981804246 abstract "Atherosclerosis is a chronic inflammatory disorder characterised by the accumulation of monocytes/macrophages, smooth muscle cells, and lymphocytes within the arterial wall in response to the release of proinflammatory molecules. Such accumulation results in the formation of the atherosclerotic plaque, which would eventually evolve to complications such as total artery occlusion, rupture, calcification, or aneurysm. Although the molecular mechanism responsible for the development of atherosclerosis is not completely understood, it is clear that the immune system plays a key role in the development of the atherosclerotic plaque and in its complications. There are multiple antigenic stimuli that have been associated with the pathogenesis of atherosclerosis. Most of these stimuli come from modified self-molecules such as oxidised low-density lipoproteins (oxLDLs), beta2glycoprotein1 (2GP1), lipoprotein a (LP(a)), heat shock proteins (HSPs), and protein components of the extracellular matrix such as collagen and fibrinogen in the form of advanced glycation-end (AGE) products. In addition, several foreign antigens including bacteria such as Porphyromonas gingivalis and Chlamydia pneumoniae and viruses such as enterovirus and cytomegalovirus have been associated with atherosclerosis as potentially causative or bystander participants, adding another level of complexity to the analysis of the pathophysiology of atherosclerosis. The present review summarises the most important scientific findings published within the last two decades on the importance of antigens, antigen stimulation, and adaptive immune responses in the development of atherosclerotic plaques." @default.
- W1981804246 created "2016-06-24" @default.
- W1981804246 creator A5018646421 @default.
- W1981804246 creator A5060791148 @default.
- W1981804246 creator A5067806317 @default.
- W1981804246 creator A5081790862 @default.
- W1981804246 date "2008-01-01" @default.
- W1981804246 modified "2023-10-18" @default.
- W1981804246 title "Antigen-Induced Immunomodulation in the Pathogenesis of Atherosclerosis" @default.
- W1981804246 cites W103895141 @default.
- W1981804246 cites W1527011538 @default.
- W1981804246 cites W1760442572 @default.
- W1981804246 cites W1899375067 @default.
- W1981804246 cites W1966590576 @default.
- W1981804246 cites W1971690241 @default.
- W1981804246 cites W1974936467 @default.
- W1981804246 cites W1976208588 @default.
- W1981804246 cites W1976591271 @default.
- W1981804246 cites W1977060426 @default.
- W1981804246 cites W1977109672 @default.
- W1981804246 cites W1978803029 @default.
- W1981804246 cites W1979226771 @default.
- W1981804246 cites W1979814568 @default.
- W1981804246 cites W1981730858 @default.
- W1981804246 cites W1985370989 @default.
- W1981804246 cites W1986868330 @default.
- W1981804246 cites W1987516580 @default.
- W1981804246 cites W1987847514 @default.
- W1981804246 cites W1993662108 @default.
- W1981804246 cites W1994641000 @default.
- W1981804246 cites W1994696883 @default.
- W1981804246 cites W1996512988 @default.
- W1981804246 cites W1996866382 @default.
- W1981804246 cites W1997388059 @default.
- W1981804246 cites W1999331681 @default.
- W1981804246 cites W1999742425 @default.
- W1981804246 cites W2002812554 @default.
- W1981804246 cites W2003906544 @default.
- W1981804246 cites W2008469419 @default.
- W1981804246 cites W2013925870 @default.
- W1981804246 cites W2016116797 @default.
- W1981804246 cites W2017622174 @default.
- W1981804246 cites W2018475901 @default.
- W1981804246 cites W2018860447 @default.
- W1981804246 cites W2019678527 @default.
- W1981804246 cites W2020057267 @default.
- W1981804246 cites W2020491804 @default.
- W1981804246 cites W2020684223 @default.
- W1981804246 cites W2022155347 @default.
- W1981804246 cites W2022397729 @default.
- W1981804246 cites W2023820547 @default.
- W1981804246 cites W2024070385 @default.
- W1981804246 cites W2025447518 @default.
- W1981804246 cites W2028859140 @default.
- W1981804246 cites W2030000356 @default.
- W1981804246 cites W2030237203 @default.
- W1981804246 cites W2031389689 @default.
- W1981804246 cites W2032207767 @default.
- W1981804246 cites W2032272012 @default.
- W1981804246 cites W2033909414 @default.
- W1981804246 cites W2035976879 @default.
- W1981804246 cites W2037062908 @default.
- W1981804246 cites W2039467806 @default.
- W1981804246 cites W2040997863 @default.
- W1981804246 cites W2045180319 @default.
- W1981804246 cites W2047353964 @default.
- W1981804246 cites W2048904988 @default.
- W1981804246 cites W2049200607 @default.
- W1981804246 cites W2050648683 @default.
- W1981804246 cites W2051653575 @default.
- W1981804246 cites W2052120337 @default.
- W1981804246 cites W2052911190 @default.
- W1981804246 cites W2054284944 @default.
- W1981804246 cites W2056682576 @default.
- W1981804246 cites W2056958787 @default.
- W1981804246 cites W2057978341 @default.
- W1981804246 cites W2058147714 @default.
- W1981804246 cites W2059514749 @default.
- W1981804246 cites W2060254028 @default.
- W1981804246 cites W2060544158 @default.
- W1981804246 cites W2061203889 @default.
- W1981804246 cites W2068378145 @default.
- W1981804246 cites W2068578933 @default.
- W1981804246 cites W2071613661 @default.
- W1981804246 cites W2072476641 @default.
- W1981804246 cites W2075185537 @default.
- W1981804246 cites W2078378682 @default.
- W1981804246 cites W2080367144 @default.
- W1981804246 cites W2081717141 @default.
- W1981804246 cites W2081850072 @default.
- W1981804246 cites W2082229626 @default.
- W1981804246 cites W2082498032 @default.
- W1981804246 cites W2086431846 @default.
- W1981804246 cites W2088889374 @default.
- W1981804246 cites W2089917722 @default.
- W1981804246 cites W2091173515 @default.
- W1981804246 cites W2092032357 @default.
- W1981804246 cites W2092112301 @default.