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• W1981937032 startingPage "1167" @default.
• W1981937032 abstract "Interleukin-10 (IL-10) is a multifunctional cytokine with diverse effects on most hematopoietic cell types. It appears the principal function of IL-10 is to limit and ultimately terminate inflammatory response. We demonstrate here that interferon-alpha2b (IFN-alpha) increases the expression of IL-10 in activated primary CD8(+) T cells. Optimal induction of mRNA expression and protein synthesis was observed when IFN-alpha was added to cells activated by the combination of anti-CD3 monoclonal antibody (mAb) and IL-2. Maximal stimulation of IL-10 protein production was observed after prolonged incubation periods (48-72 h). No effects were observed on the production of IL-4, whereas IFN-gamma was produced with a faster kinetics than an untreated control. Our data indicate that IFN-alpha promotes the development of a CD8(+) T cell population with enhanced anti-inflammatory activity, which may play a critical role in the regulation of a proper immune response." @default.
• W1981937032 created "2016-06-24" @default.
• W1981937032 creator A5031691457 @default.
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• W1981937032 date "2002-12-01" @default.
• W1981937032 modified "2023-09-23" @default.
• W1981937032 title "IFN-<i>α</i>2b Increases Interleukin-10 Expression in Primary Activated Human CD8⁺T Cells" @default.
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• W1981937032 doi "https://doi.org/10.1089/10799900260475678" @default.
• W1981937032 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/12581488" @default.
• W1981937032 hasPublicationYear "2002" @default.
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