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- W1982141574 endingPage "373" @default.
- W1982141574 startingPage "361" @default.
- W1982141574 abstract "The most profound biochemical phenotype of cancer cells is their ability to metabolize glucose to lactate, even under aerobic conditions. This alternative metabolic circuitry is sufficient to support the biosynthetic and energy requirements for cancer cell proliferation and metastasis. Alterations in oncogenes and tumor-suppressor genes are involved in the metabolic switch of cancer cells to aerobic glycolysis, increased glutaminolysis, and fatty acid biosynthesis. miRNAs mediate fine-tuning of genes involved directly or indirectly in cancer metabolism. In this review we discuss the regulatory role of miRNAs on enzymes, signaling pathways, and transcription factors involved in glucose and lipid metabolism. We further consider the therapeutic potential of metabolism-related miRNAs in cancer." @default.
- W1982141574 created "2016-06-24" @default.
- W1982141574 creator A5026535780 @default.
- W1982141574 creator A5049770365 @default.
- W1982141574 creator A5064972382 @default.
- W1982141574 date "2013-07-01" @default.
- W1982141574 modified "2023-10-18" @default.
- W1982141574 title "miRNAs link metabolic reprogramming to oncogenesis" @default.
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