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- W1982243818 abstract "Problem Differences in the expression of gp96 and its receptors were analysed in normal and pathological human pregnancy. Material and Methods Immunohistology and immunofluorescence of sections from decidual part of term placenta, first trimester normal decidua, missed abortion and blighted ovum decidua were performed together with reverse transcriptase–quantitative polymerase chain reaction and flow cytometry. Results In missed abortion, gp96 was intensively stained, when compared to normal early pregnancy. The intensity of CD 91 and TLR 4 was higher in the first trimester pregnancy and blighted ovum, when compared to missed abortion. Decidual part of the term placenta is invaded with gp96 + , CD 91 + and TLR 4+ trophoblast. Progesterone‐induced blocking factor ( PIBF ) decreased the frequency of TLR 4 + T lymphocytes, CD 91 + T , natural killer ( NK ) and mature dendritic cells after an 18‐h culture. Decidual mononuclear cells ( DMC s) treated with PIBF down‐regulated CD 91, TLR 4 and gp96 gene expression. Conclusion The presence of gp96, CD 91 and TLR 4 at the maternal–foetal interface provides a molecular basis for their interaction, particularly in the absence of PIBF ." @default.
- W1982243818 created "2016-06-24" @default.
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- W1982243818 date "2013-06-11" @default.
- W1982243818 modified "2023-10-17" @default.
- W1982243818 title "The Significance of Heat-Shock Protein GP96 and its Receptors' CD91 and Toll-Like Receptor 4 Expression at the Maternal Foetal Interface" @default.
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- W1982243818 doi "https://doi.org/10.1111/aji.12096" @default.
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