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• W1983497985 abstract "Experimental autoimmune encephalomyelitis (EAE) is a widely adopted animal model system for studying human multiple sclerosis that affects the central nervous system (CNS). To understand the underlying pathogenic mechanisms of the autoimmune T cell response, localization, enumeration and characterization of autoreactive T cells are essential. We assessed encephalitogenic proteolipid protein epitope (PLP139–151)-specific T cells in the periphery and CNS of SJL/J mice using MHC class II I-As multimers during both pre-clinical and clinical phases of PLP-induced EAE in conjunction with T cell function. Our results strongly suggest that PLP139–151-specific CD4+ T cells first expand primarily in the CNS-draining cervical lymph nodes and then migrate to the CNS. In the CNS, these PLP-specific CD4+ T cells accumulate, become activated and differentiate into effector cells that produce IFN-γ in response to the self-peptide." @default.
• W1983497985 created "2016-06-24" @default.
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• W1983497985 date "2004-10-01" @default.
• W1983497985 modified "2023-09-29" @default.
• W1983497985 title "Functional maturation of proteolipid protein139–151-specific Th1 cells in the central nervous system in experimental autoimmune encephalomyelitis" @default.
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• W1983497985 doi "https://doi.org/10.1016/j.jneuroim.2004.06.012" @default.
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