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- W1983521522 endingPage "793" @default.
- W1983521522 startingPage "777" @default.
- W1983521522 abstract "The heart has complex mechanisms that facilitate the maintenance of an oxygen supply–demand balance necessary for its contractile function in response to physiological fluctuations in workload as well as in response to chronic stresses such as hypoxia, ischemia, and overload. Redox-sensitive signaling pathways are centrally involved in many of these homeostatic and stress-response mechanisms. Here, we review the main redox-regulated pathways that are involved in cardiac myocyte excitation–contraction coupling, differentiation, hypertrophy, and stress responses. We discuss specific sources of endogenously generated reactive oxygen species (e.g., mitochondria and NADPH oxidases of the Nox family), the particular pathways and processes that they affect, the role of modulators such as thioredoxin, and the specific molecular mechanisms that are involved—where this knowledge is available. A better understanding of this complex regulatory system may allow the development of more specific therapeutic strategies for heart diseases." @default.
- W1983521522 created "2016-06-24" @default.
- W1983521522 creator A5019437338 @default.
- W1983521522 creator A5060342135 @default.
- W1983521522 creator A5080438350 @default.
- W1983521522 creator A5084583713 @default.
- W1983521522 creator A5084971249 @default.
- W1983521522 date "2011-04-01" @default.
- W1983521522 modified "2023-10-12" @default.
- W1983521522 title "Redox signaling in cardiac myocytes" @default.
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