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• W1983754037 abstract "Abstract: In the isolated guinea-pig bronchus, the potassium channel blocking agent 4-aminopyridine (10-4 M) caused a contraction which was abolished by capsaicin tachyphylaxis, suggesting involvement of sensory neuropeptides. Charybdotoxin (10-8, 5 × 10-8 M), which is a potent blocker of the high-conductance Ca2+-activated K+ channel in smooth muscle, caused slowly developing and long lasting bronchoconstriction, which was resistant to capsaicin tachyphylaxis. Neither 4-aminopyridine (10-3, 10-4 M) nor charybdotoxin (10-8, 5 × 10-8 M) had any significant effect on the bronchoconstriction induced by electrical field stimulation. Furthermore, charybdotoxin had no significant influence on the inhibitory effect of the α2-adrenoceptor agonist SKF 35886 (5 × 10-7 M) on the bronchoconstriction induced by electrical field stimulation. In the isolated perfused guinea-pig lung, 4-aminopyridine (3 × 10-5- 10-3 M) caused bronchoconstriction and enhanced both basal and (at 3 × 10-5 M) vagal nerve stimulation-evoked calcitonin gene-related peptide outflow from pulmonary sensory nerves. In conclusion, 4-aminopyridine stimulated capsaicin-sensitive sensory neurons and enhanced the sensory activation induced by vagal nerve stimulation in guinea-pig lung. Charybdotoxin, on the other hand, caused bronchial contraction independently of capsaicin-sensitive nerves." @default.
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• W1983754037 date "1993-02-01" @default.
• W1983754037 modified "2023-10-18" @default.
• W1983754037 title "Different Effects of the K⁺Channel Blockers 4-Aminopyridine and Charybdotoxin on Sensory Nerves in Guinea-pig Lung" @default.
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• W1983754037 doi "https://doi.org/10.1111/j.1600-0773.1993.tb00305.x" @default.
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