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- W1984189107 abstract "In the present study, we evaluated the ability of 4-hydroxytamoxifen (OHT) and epidermal growth factor (EGF) to regulate homotypic adhesion in MCF7 breast cancer cells. Our results demonstrate that OHT and EGF activate the E-cadherin promoter, increase E-cadherin mRNA and protein expression and enhance homotypic aggregation of MCF7 cells. Interestingly, an ERα and EGFR cross-talk is involved in the E-cadherin expression by OHT and EGF, as demonstrated by knocking down either receptor. On the basis of our findings, the well-established cross-talk between ERα and EGFR could be extended to the modulation of E-cadherin expression by OHT and EGF. Thus, the potential ability of tamoxifen to induce cell–cell aggregation may contribute to the biologic response of pharmacologic intervention in patients with breast cancer." @default.
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- W1984189107 date "2009-07-11" @default.
- W1984189107 modified "2023-09-29" @default.
- W1984189107 title "E-cadherin mediates the aggregation of breast cancer cells induced by tamoxifen and epidermal growth factor" @default.
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- W1984189107 doi "https://doi.org/10.1007/s10549-009-0456-4" @default.
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