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• W1984270332 endingPage "682" @default.
• W1984270332 startingPage "677" @default.
• W1984270332 abstract "Transglutaminase 2 (tissue transglutaminase, TGase 2) was recently identified as an endomysial autoantigen in celiac disease (CD). Identification of how TGase 2 expression is increased may allow a better understanding of this autoimmune disease. Certain inflammatory cytokines, tumor necrosis factor-alpha (TNF-alpha) and transforming growth factor-beta (TGF-beta), and the Th type I cytokine interferon-gamma (INF-gamma) are abundant in CD. We have investigated whether these play a role in the regulation of TGase 2 expression in a model rat small intestinal epithelial cell line (IEC-6). After treatment for 24 h, TNF-alpha did not significantly alter TGase 2 mRNA or activity, but TGF-beta decreased mRNA and activity by 4-5-fold. IFN-gamma increased mRNA and TGase 2 activity by about 2-fold in 24 h and 5-fold by 5 days. Our new data suggest that increased TGase 2 expression in the upper small intestine of CD patients may be due to increased IFN-gamma expression, loss of TGF-beta signaling, or both." @default.
• W1984270332 created "2016-06-24" @default.
• W1984270332 creator A5014464546 @default.
• W1984270332 creator A5016276414 @default.
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• W1984270332 date "2002-06-01" @default.
• W1984270332 modified "2023-10-18" @default.
• W1984270332 title "IFN-<i>γ</i>Induces Transglutaminase 2 Expression in Rat Small Intestinal Cells" @default.
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• W1984270332 doi "https://doi.org/10.1089/10799900260100169" @default.
• W1984270332 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/12162878" @default.
• W1984270332 hasPublicationYear "2002" @default.
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