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- W1984723092 abstract "Free oxygen radicals generated by anthracycline/iron complexes have been implicated in anthracycline cytotoxicity. We therefore tested whether enzymatic scavengers of free radicals or metal chelators were able to inhibit anthracycline toxicity. The survival of Chinese hamster fibroblasts was reduced when the cells were exposed to 0.1 – 1.0 mg/l 4′-epidoxorubicin (epirubicin). Superoxide dismutase (SOD) (250 mg/l), or catalase (250 mg/l) did not affect the clonogenic survival of the fibroblasts. The metal-chelators, diethylenetriamine-pentacetic acid (DTPA) (100 μmol/l), EDTA (100 μmol/l), and desferrixamine (100 μmol/l) all protected against epirubicin-induced clonogenic survival. The protection of chelators against epirubicin toxicity implies that chelators might also be able to modulate anthracycline toxicity in vivo." @default.
- W1984723092 created "2016-06-24" @default.
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- W1984723092 date "1988-12-01" @default.
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- W1984723092 title "Protective effect of iron chelators on epirubicin-induced fibroblast toxicity" @default.
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- W1984723092 doi "https://doi.org/10.1016/0304-3835(88)90168-1" @default.
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