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• W1985323765 endingPage "1027" @default.
• W1985323765 startingPage "1022" @default.
• W1985323765 abstract "We determined relationships of the anti-oxidative enzyme, paraoxonase-1 (PON-1), with high density lipoprotein (HDL) subfractions, and tested whether these relationships are stronger than those with HDL cholesterol and apolipoprotein A-I (apoA-I) in subjects with and without type 2 diabetes mellitus (T2DM). Serum PON-1 (arylesterase activity) and HDL subfractions (nuclear magnetic resonance spectroscopy) were determined in 67 T2DM patients and in 56 non-diabetic subjects. PON-1 activity, HDL cholesterol and apoA-I were decreased in T2DM (all p < 0.05). The HDL particle concentration was unaltered, but large HDL particles, medium HDL particles and HDL particle size were decreased, whereas small HDL particles were increased in T2DM (all p < 0.05). PON-1 was more closely related to HDL cholesterol than to apoA-I (p = 0.001). In turn, the positive relationship of PON-1 with the HDL particle concentration and with large HDL particles was stronger than that with HDL cholesterol (both p < 0.01). The inverse relationship of PON-1 with T2DM was only modestly attenuated by HDL cholesterol or HDL particle characteristics. PON-1 activity is more closely related to the HDL particle concentration or large HDL particles than to HDL cholesterol. Impaired PON-1 activity in T2DM is not to a considerable extent explained by altered HDL subfraction levels." @default.
• W1985323765 created "2016-06-24" @default.
• W1985323765 creator A5059663377 @default.
• W1985323765 creator A5063490617 @default.
• W1985323765 creator A5069795479 @default.
• W1985323765 date "2014-08-01" @default.
• W1985323765 modified "2023-10-02" @default.
• W1985323765 title "Serum paraoxonase-1 activity is more closely related to HDL particle concentration and large HDL particles than to HDL cholesterol in Type 2 diabetic and non-diabetic subjects" @default.
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• W1985323765 doi "https://doi.org/10.1016/j.clinbiochem.2014.04.013" @default.
• W1985323765 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/24769273" @default.
• W1985323765 hasPublicationYear "2014" @default.
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