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• W1985364936 endingPage "IJTR.S11355" @default.
• W1985364936 startingPage "IJTR.S11355" @default.
• W1985364936 abstract "Autism spectrum disorder (ASD) is a pervasive neuro-developmental disorder characterized by impaired social interaction, reduced/absent verbal and non-verbal communication, and repetitive behavior during early childhood. The etiology of this developmental disorder is poorly understood, and no biomarkers have been identified. Identification of novel biochemical markers related to autism would be advantageous for earlier clinical diagnosis and intervention. Studies suggest that oxidative stress-induced mechanisms and reduced antioxidant defense, mitochondrial dysfunction, and impaired energy metabolism (NAD + , NADH, ATP, pyruvate, and lactate), are major causes of ASD. This review provides renewed insight regarding current autism research related to oxidative stress, mitochondrial dysfunction, and altered tryptophan metabolism in ASD." @default.
• W1985364936 created "2016-06-24" @default.
• W1985364936 creator A5000040292 @default.
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• W1985364936 creator A5052925147 @default.
• W1985364936 creator A5071713965 @default.
• W1985364936 creator A5075562396 @default.
• W1985364936 date "2013-01-01" @default.
• W1985364936 modified "2023-10-16" @default.
• W1985364936 title "Role of NAD⁺, Oxidative Stress, and Tryptophan Metabolism in Autism Spectrum Disorders" @default.
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