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- W1985460395 endingPage "730" @default.
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- W1985460395 abstract "The neuromuscular junction (NMJ) is a synapse that develops between a motor neuron and a muscle fiber. A defining feature of NMJ development in vertebrates is the re-distribution of muscle acetylcholine (ACh) receptors (AChRs) following innervation, which generates high-density AChR clusters at the postsynaptic membrane and disperses aneural AChR clusters formed in muscle before innervation. This process in vivo requires MuSK, a muscle-specific receptor tyrosine kinase that triggers AChR re-distribution when activated; rapsyn, a muscle protein that binds and clusters AChRs; agrin, a nerve-secreted heparan-sulfate proteoglycan that activates MuSK; and ACh, a neurotransmitter that stimulates muscle and also disperses aneural AChR clusters. Moreover, in cultured muscle cells, several additional muscle- and nerve-derived molecules induce, mediate or participate in AChR clustering and dispersal. In this review we discuss how regulation of AChR re-distribution by multiple factors ensures aggregation of AChRs exclusively at NMJs. IUBMB Life, 57: 719-730, 2005" @default.
- W1985460395 created "2016-06-24" @default.
- W1985460395 creator A5009104784 @default.
- W1985460395 creator A5023468497 @default.
- W1985460395 date "2005-11-01" @default.
- W1985460395 modified "2023-10-12" @default.
- W1985460395 title "Molecular regulation of postsynaptic differentiation at the neuromuscular junction" @default.
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