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• W1985493669 abstract "Animal models of human brain damage syndromes range from ones that are purely theoretical to ones that attempt to duplicate a human syndrome at every level. The similarity between animal and human cases in experimental manipulation, CNS alteration, and tests of parallelism is sometimes restricted by the extent to which experimental species match humans in their basic biology. However, the information available from the human condition is often the most important limiting factor in the degree to which similarities can be achieved. The recent discovery of a high rate of autism in patients exposed to thalidomide around the time of neural tube closure suggests an appropriate experimental manipulation for an animal model of the disease. Rats exposed to valproic acid at the same stage of development mimic the thalidomide cases' apparent injuries to the brain stem. Whether the behavior of these brain-damaged animals is similar to that observed in autism is difficult to determine, because the diagnostic behavioral features of the disease are not easily translated into animal tasks. However, anatomical observations in human cases provide tests of parallelism that can be applied to animals, and these appear to be positive in the animal model. The development of behavioral tests that discriminate “autistic behavior” from that of other brain damage syndromes may require further research in both humans and animals. © 1997 Wiley-Liss, Inc." @default.
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• W1985493669 date "1996-01-01" @default.
• W1985493669 modified "2023-09-25" @default.
• W1985493669 title "Animal model of autism based on developmental data" @default.
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• W1985493669 doi "https://doi.org/10.1002/(sici)1098-2779(1996)2:4<249::aid-mrdd9>3.0.co;2-l" @default.
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