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- W1986032412 abstract "CFTR is a cAMP-activated chloride and bicarbonate channel that is critical for lung homeostasis. Decreases in CFTR expression have dire consequences in cystic fibrosis (CF) and have been suggested to be a component of the lung pathology in chronic obstructive pulmonary disease. Decreases or loss of channel function often lead to mucus stasis, chronic bacterial infections, and the accompanying chronic inflammatory responses that promote progressive lung destruction, and, eventually in CF, lung failure. Here we discuss CFTR's functional role airway surface liquid hydration and pH, in regulation of other channels such as the epithelial sodium channel, and in regulating inflammatory responses in the lung." @default.
- W1986032412 created "2016-06-24" @default.
- W1986032412 creator A5039708431 @default.
- W1986032412 creator A5090337748 @default.
- W1986032412 date "2014-12-15" @default.
- W1986032412 modified "2023-10-05" @default.
- W1986032412 title "CFTR and lung homeostasis" @default.
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- W1986032412 doi "https://doi.org/10.1152/ajplung.00326.2014" @default.
- W1986032412 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/4269691" @default.
- W1986032412 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/25381027" @default.
- W1986032412 hasPublicationYear "2014" @default.
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