FRINK Linked Data Fragments server

Matches in SemOpenAlex for { <https://semopenalex.org/work/W1986287343> ?p ?o ?g. }

• W1986287343 endingPage "757" @default.
• W1986287343 startingPage "738" @default.
• W1986287343 abstract "Mycotoxins are produced mainly by the mycelial structure of filamentous fungi, or more specifically, molds. These secondary metabolites are synthesized during the end of the exponential growth phase and appear to have no biochemical significance in fungal growth and development. The contamination of foods and feeds with mycotoxins is a significant problem for the adverse effects on humans, animals, and crops that result in illnesses and economic losses. The toxic effect of the ingestion of mycotoxins in humans and animals depends on a number of factors including intake levels, duration of exposure, toxin species, mechanisms of action, metabolism, and defense mechanisms. In general, the consumption of contaminated food and feed with mycotoxin induces to neurotoxic, immunosuppressive, teratogenic, mutagenic, and carcinogenic effect in humans and/or animals. The most significant mycotoxins in terms of public health and agronomic perspective include the aflatoxins, ochratoxin A (OTA), trichothecenes, fumonisins, patulin, and the ergot alkaloids. Due to the detrimental effects of these mycotoxins, several strategies have been developed in order to reduce the risk of exposure. These include the degradation, destruction, inactivation or removal of mycotoxins through chemical, physical and biological methods. However, the results obtained with these methods have not been optimal, because they may change the organoleptic characteristics and nutritional values of food. Another alternative strategy to prevent or reduce the toxic effects of mycotoxins is by applying antimutagenic agents. These substances act according to several extra- or intracellular mechanisms, their main goal being to avoid the interaction of mycotoxins with DNA; as a consequence of their action, these agents would inhibit mutagenesis and carcinogenesis. This article reviews the main strategies used to control AFB1 and ochratoxin A and contains an analysis of some antigenotoxic substances that reduce the DNA damage caused by these mycotoxins." @default.
• W1986287343 created "2016-06-24" @default.
• W1986287343 creator A5015784838 @default.
• W1986287343 creator A5033484262 @default.
• W1986287343 creator A5040433584 @default.
• W1986287343 creator A5054318343 @default.
• W1986287343 creator A5055588585 @default.
• W1986287343 creator A5065367333 @default.
• W1986287343 creator A5068803276 @default.
• W1986287343 creator A5091525533 @default.
• W1986287343 date "2010-04-19" @default.
• W1986287343 modified "2023-10-16" @default.
• W1986287343 title "Antigenotoxic Studies of Different Substances to Reduce the DNA Damage Induced by Aflatoxin B1 and Ochratoxin A" @default.
• W1986287343 cites W1213650891 @default.
• W1986287343 cites W1544825355 @default.
• W1986287343 cites W1967935247 @default.
• W1986287343 cites W1968654649 @default.
• W1986287343 cites W1968889897 @default.
• W1986287343 cites W1969509823 @default.
• W1986287343 cites W1971210013 @default.
• W1986287343 cites W1973986846 @default.
• W1986287343 cites W1976226813 @default.
• W1986287343 cites W1976578900 @default.
• W1986287343 cites W1977089846 @default.
• W1986287343 cites W1981990436 @default.
• W1986287343 cites W1984358216 @default.
• W1986287343 cites W1984485111 @default.
• W1986287343 cites W1985635433 @default.
• W1986287343 cites W1986810464 @default.
• W1986287343 cites W1988426570 @default.
• W1986287343 cites W1994409904 @default.
• W1986287343 cites W1995190878 @default.
• W1986287343 cites W2001777724 @default.
• W1986287343 cites W2003729646 @default.
• W1986287343 cites W2005451967 @default.
• W1986287343 cites W2005527209 @default.
• W1986287343 cites W2006666559 @default.
• W1986287343 cites W2012054966 @default.
• W1986287343 cites W2016221744 @default.
• W1986287343 cites W2016274911 @default.
• W1986287343 cites W2020156245 @default.
• W1986287343 cites W2022976246 @default.
• W1986287343 cites W2024095876 @default.
• W1986287343 cites W2028189744 @default.
• W1986287343 cites W2032698170 @default.
• W1986287343 cites W2034848149 @default.
• W1986287343 cites W2040474418 @default.
• W1986287343 cites W2043051127 @default.
• W1986287343 cites W2049185204 @default.
• W1986287343 cites W2050150328 @default.
• W1986287343 cites W2050154615 @default.
• W1986287343 cites W2055864750 @default.
• W1986287343 cites W2057494515 @default.
• W1986287343 cites W2058112167 @default.
• W1986287343 cites W2058514558 @default.
• W1986287343 cites W2063185576 @default.
• W1986287343 cites W2065560805 @default.
• W1986287343 cites W2074672869 @default.
• W1986287343 cites W2075445024 @default.
• W1986287343 cites W2076153589 @default.
• W1986287343 cites W2079360541 @default.
• W1986287343 cites W2080828655 @default.
• W1986287343 cites W2083392737 @default.
• W1986287343 cites W2085046967 @default.
• W1986287343 cites W2089301511 @default.
• W1986287343 cites W2089698923 @default.
• W1986287343 cites W2103178225 @default.
• W1986287343 cites W2116030627 @default.
• W1986287343 cites W2117849826 @default.
• W1986287343 cites W2121364257 @default.
• W1986287343 cites W2121829036 @default.
• W1986287343 cites W2123087385 @default.
• W1986287343 cites W2125215178 @default.
• W1986287343 cites W2126902508 @default.
• W1986287343 cites W2155636403 @default.
• W1986287343 cites W2156478120 @default.
• W1986287343 cites W2157168777 @default.
• W1986287343 cites W2158449128 @default.
• W1986287343 cites W2160746935 @default.
• W1986287343 cites W2167366085 @default.
• W1986287343 cites W2168288146 @default.
• W1986287343 cites W2179237616 @default.
• W1986287343 cites W2286069758 @default.
• W1986287343 cites W2559424949 @default.
• W1986287343 cites W4211237594 @default.
• W1986287343 doi "https://doi.org/10.3390/toxins2040738" @default.
• W1986287343 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3153197" @default.
• W1986287343 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/22069607" @default.
• W1986287343 hasPublicationYear "2010" @default.
• W1986287343 type Work @default.
• W1986287343 sameAs 1986287343 @default.
• W1986287343 citedByCount "29" @default.
• W1986287343 countsByYear W19862873432012 @default.
• W1986287343 countsByYear W19862873432013 @default.
• W1986287343 countsByYear W19862873432014 @default.
• W1986287343 countsByYear W19862873432015 @default.
• W1986287343 countsByYear W19862873432016 @default.
• W1986287343 countsByYear W19862873432017 @default.

• next