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• W1987194328 abstract "The pathophysiology of depression remains enigmatic, although abnormalities in serotonin signaling have been implicated. We have found that the serotonin 1B receptor [5-hydroxytryptamine (5-HT 1B ) receptor] interacts with p11. p11 increases localization of 5-HT 1B receptors at the cell surface. p11 is increased in rodent brains by antidepressants or electroconvulsive therapy, but decreased in an animal model of depression and in brain tissue from depressed patients. Overexpression of p11 increases 5-HT 1B receptor function in cells and recapitulates certain behaviors seen after antidepressant treatment in mice. p11 knockout mice exhibit a depression-like phenotype and have reduced responsiveness to 5-HT 1B receptor agonists and reduced behavioral reactions to an antidepressant." @default.
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• W1987194328 date "2006-01-06" @default.
• W1987194328 modified "2023-10-16" @default.
• W1987194328 title "Alterations in 5-HT _1B Receptor Function by p11 in Depression-Like States" @default.
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• W1987194328 doi "https://doi.org/10.1126/science.1117571" @default.
• W1987194328 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/16400147" @default.
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