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- W1987474016 abstract "We have recently observed that anemic and dystrophic monkeys develop reticulocytosis on treatment with a normally synthesized body component, coenzyme Q10 (Fitch, et al., 1965). Complete remission of the anemia is effected by hexahydrocoenzyme Q4 therapy (Fitch, et al., 1965). The anemia, which develops in monkeys deprived of vitamin E, also responds well to vitamin E-treatment (Dinning and Day, 1957). This apparent connection between an intrinsic coenzyme and a vitamin has been studied by observing the ability of hexahydrocoenzyme Q4 to protect red blood cells from lysis by H2O2. This study was prompted by two facts: (a) vitamin E protects cells and subcellular particles from lysis by oxidants (Rose and Győrgy, 1952; McKnight, Hunter, and Oehlert, 1965), and (b) coenzyme Q is present in the lipoprotein complexes of cellular membranes (Green, 1965). We found that hexahydrocoenzyme Q4 and the 6-chromanol of hexahydrocoenzyme Q4 protect erythrocytes of premature infants from hemolysis by H2O2." @default.
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- W1987474016 date "1967-01-01" @default.
- W1987474016 modified "2023-09-26" @default.
- W1987474016 title "Coenzyme Q and the stability of biological membranes" @default.
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- W1987474016 doi "https://doi.org/10.1016/0006-291x(67)90223-9" @default.
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