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• W1989492087 endingPage "526" @default.
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• W1989492087 abstract "The Arabidopsis accelerated cell death 6-1 (acd6-1) mutant shows constitutive defense, cell death, and extreme dwarf phenotypes. In a screen for acd6-1 suppressors, we identified a mutant that was disrupted by a T-DNA in the PHOSPHATE TRANSPORTER 4;1 (PHT4;1) gene. The suppressor mutant pht4;1-1 is dominant, expresses truncated PHT4;1 transcripts, and is more susceptible to virulent Pseudomonas syringae strains but not to several avirulent strains. Treatment with a salicylic acid (SA) agonist induced a similar level of resistance in Col-0 and pht4;1-1, suggesting that PHT4;1 acts upstream of the SA pathway. Genetic analysis further indicates that PHT4;1 contributes to SID2-dependent and -independent pathways. Transgenic expression of the DNA fragment containing the PHT4;1-1 region or the full-length PHT4;1 gene in wild-type conferred enhanced susceptibility to Pseudomonas infection. Interestingly, expression of PHT4;1 is regulated by the circadian clock. Together, these data suggest that the phosphate transporter PHT4;1 is critical for basal defense and also implicate a potential role of the circadian clock in regulating innate immunity of Arabidopsis." @default.
• W1989492087 created "2016-06-24" @default.
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• W1989492087 date "2011-05-01" @default.
• W1989492087 modified "2023-10-17" @default.
• W1989492087 title "Circadian Clock-Regulated Phosphate Transporter PHT4;1 Plays an Important Role in Arabidopsis Defense" @default.
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• W1989492087 doi "https://doi.org/10.1093/mp/ssr016" @default.
• W1989492087 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3988428" @default.
• W1989492087 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/21447757" @default.
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