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• W1989754265 abstract "SUMMARY Inflammatory breast cancer (IBC) is a very aggressive form of breast cancer with a high mortality rate. Most patients have lymph node metastasis at the time of diagnosis and 30% of patients already have metastases in distant organs. IBC is normally treated with multimodality therapy. Endocrine therapy is administered in cases of ER-positive tumors. Nevertheless, IBC has a high HOXB13:Il17RB ratio, predicting a poor response to tamoxifen treatment. These data suggest a possible role for IBC as a model for endocrine resistance. Previous studies have shown that NF-κB, a transcription factor regulating different cellular processes, is more highly activated in IBC than in non-IBC, while ER is often downregulated in this tumor type. This article summarizes the activity of ER and NF-κB in IBC and their possible contribution to endocrine resistance in this breast cancer subtype." @default.
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• W1989754265 date "2014-01-01" @default.
• W1989754265 modified "2023-10-16" @default.
• W1989754265 title "Contribution of ER and NF-κB to endocrine resistance in inflammatory breast cancer" @default.
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• W1989754265 doi "https://doi.org/10.2217/bmt.13.72" @default.
• W1989754265 hasPublicationYear "2014" @default.
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