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• W1989868421 abstract "Abstract Four cases of jaundice are described in which the hyperbilirubinemia was not related to the breakdown of circulating erythrocytes or to a defect in hepatic function. Three of the cases described occurred in one family and are thought to represent a congenital abnormality of excessive bilirubin production from sources other than the circulating red cell. This primary overproduction of bilirubin has been termed shunt hyperbilirubinemia. The hyperbilirubinemia was characterized by the onset of jaundice and splenomegaly in early adult life. The serum bilirubin was predominantly indirect reacting pigment, and the red cell survival somewhat short prior to splenectomy. The urobilinogen excretion in urine and stool was high. Hemolysis, when present, was corrected by splenectomy but the reticulocytosis, normoblastic hyperplasia of the marrow, hyperbilirubinemia and increased urobilinogen excretion persisted. The liver showed gross hemosiderin deposits in the parenchymal cells in the periphery of the lobule. It is suggested that the excess bilirubin has as its source the marrow erythrocytes or their precursors, either by their destruction or their synthesis of bilirubin directly from heme or its tetrapyrrole antecedents." @default.
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• W1989868421 date "1959-11-01" @default.
• W1989868421 modified "2023-09-26" @default.
• W1989868421 title "Hyperbilirubinemia due to an alternate path of bilirubin production" @default.
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• W1989868421 doi "https://doi.org/10.1016/0002-9343(59)90186-x" @default.
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