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• W1990244564 abstract "Hepatocyte growth factor (HGF) and its receptor, the Met tyrosine kinase, are determinants of placenta, liver, and muscle development. Here, we show that Met function in vivo requires signaling via two carboxy-terminal tyrosines. Mutation of both residues in the mouse genome caused embryonal death, with placenta, liver, and limb muscle defects, mimicking the phenotype of met null mutants. In contrast, disrupting the consensus for Grb2 binding allowed development to proceed to term without affecting placenta and liver but caused a striking reduction in limb muscle coupled to a generalized deficit of secondary fibers. These data show that the requirements for Met signaling vary depending on the tissue and reveal a novel role for HGF/Met in late myogenesis." @default.
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• W1990244564 date "1996-11-01" @default.
• W1990244564 modified "2023-10-11" @default.
• W1990244564 title "Uncoupling of Grb2 from the Met Receptor In Vivo Reveals Complex Roles in Muscle Development" @default.
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• W1990244564 doi "https://doi.org/10.1016/s0092-8674(00)81372-0" @default.
• W1990244564 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/8898205" @default.
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