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• W1990257265 abstract "The role of glial cells in nitric oxide production in the cerebellum of conscious rats was investigated with a glial selective metabolic inhibitor, fluorocitrate. The levels of nitric oxide metabolites (nitrite plus nitrate) in the dialysate following in vivo microdialysis progressively increased to more than 2-fold the basal levels during a 2-h infusion of fluorocitrate (1 mM), and the increase persisted for more than 2 h after the treatment. Pretreatment with NG-nitro-l-arginine methyl ester attenuated the fluorocitrate-induced increase in nitric oxide metabolite levels. None of the glutamate receptor antagonists, including d(−)-2-amino-5-phosphonopentanoic acid, 6,7-dinitroquinoxaline-2,3-dione, and (±)-α-methyl-4-carboxyphenylglycine, inhibited the fluorocitrate-induced increase. The l-arginine-induced increase was significantly reduced by fluorocitrate treatment, while N-methyl-d-aspartate, (+)-α-amino-3-hydroxy-5-methylisoxazole-4-propionic acid, and trans-(±)-1-amino-(1S,3R)-cyclopentane-dicarboxylic acid increased nitric oxide metabolites levels in the fluorocitrate-treated rats, as much as in control animals. These results suggest that glial cells play an important role in modulating nitric oxide production in the cerebellum by regulating l-arginine availability." @default.
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• W1990257265 date "1997-07-01" @default.
• W1990257265 modified "2023-09-26" @default.
• W1990257265 title "Changes in extracellular nitrite and nitrate levels after inhibition of glial metabolism with fluorocitrate" @default.
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• W1990257265 doi "https://doi.org/10.1016/s0006-8993(97)00372-7" @default.
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