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• W1990266943 abstract "The cytokine tumor necrosis factor (TNF) elicits a wide range of biological responses, including inflammation, cell proliferation, differentiation, and apoptosis. Although the molecular mechanisms of TNF signaling have been largely elucidated, the principle that regulates the balance of life and death is still unknown. This review will focus on the crosstalk that exists between proteins of the TNF receptor (TNF-R) signalosome, and which are involved in the initiation of nuclear factor kappa B (NF-kappaB) activation or apoptosis. At least four different mechanisms of regulation can be distinguished: (i) NF-kappaB-mediated induction of proteins of the TNF-R complex; (ii) NF-kappaB-independent protection against apoptosis by the TNF-R-associating factor 2 (TRAF2)-mediated recruitment of antiapoptotic proteins; (iii) dual activation of apoptosis and NF-kappaB by a single molecule; and (iv) amplification of the death signal by proteolytic inactivation of signaling proteins that are involved in NF-kappaB activation or cell survival." @default.
• W1990266943 created "2016-06-24" @default.
• W1990266943 creator A5012926019 @default.
• W1990266943 creator A5080918885 @default.
• W1990266943 date "2001-09-01" @default.
• W1990266943 modified "2023-10-12" @default.
• W1990266943 title "Crosstalk between NF-κB-Activating and Apoptosis-Inducing Proteins of the TNF-Receptor Complex" @default.
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• W1990266943 doi "https://doi.org/10.1006/mcbr.2001.0295" @default.
• W1990266943 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/11529675" @default.
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