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- W1990673560 abstract "HB 699 is a non-sulfonylurea acyl-amino-alkyl benzoic acid derivative, corresponding to a major part of the glibenclamide molecule. Basal insulin release (3 mmol/l glucose) as well as glucose-induced release (10 mmol/l glucose) were stimulated by 25 mumol/l and 200 mumol/l HB 699. HB 699 (200 mumol/l) had no effect on the osmotic swelling induced by hypoosmolarity (180 mosm/l). The results indicate that the glibenclamide-induced insulin release can be resolved in a high-affinity component, which correlates with increased osmotic resistance in the beta-cells and a low-affinity component not associated with increased osmotic resistance. It is suggested that the latter component may be due to the part of the glibenclamide molecule that corresponds to HB 699." @default.
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- W1990673560 date "1984-02-01" @default.
- W1990673560 modified "2023-09-27" @default.
- W1990673560 title "The acyl-amino-alkyl benzoic acid residue and the sulfonylurea containing residue of glibenclamide affect different aspects of β-cell function" @default.
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- W1990673560 doi "https://doi.org/10.1111/j.1748-1716.1984.tb00135.x" @default.
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