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- W1991292831 abstract "The prevention of Ca(2+)-induced permeabilization of rat liver and kidney cortex mitochondria by dexamethasone, a common anti-inflammatory glucocorticoid, was the subject of this study. A non-specific release of matrix Ca2+ and membrane depolarization was observed in respiring mitochondrial suspensions subjected to a 30 nmol Ca2+/mg protein load in the presence of 2 mM inorganic phosphate (Pi), or 20 nmol Ca2+/mg protein with 1 mM Pi, for liver and renal cortex mitochondria (RCM), respectively. Additions of dexamethasone prior to Ca2+ in mitochondrial suspensions from liver or kidney cortex (80 and 200 microM final concentrations, respectively) led to 75-80% protection from these permeabilization-associated alterations of functional integrity. In conclusion, dexamethasone appears to show great promise in blocking the opening of a Ca(2+)-dependent 'non-specific pore' in the inner membranes of mitochondria from various sources." @default.
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- W1991292831 date "1995-06-01" @default.
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- W1991292831 title "Dexamethasone inhibits inorganic phosphate stimulated Ca2+-dependent damage of isolated rat liver and renal cortex mitochondria" @default.
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- W1991292831 doi "https://doi.org/10.1016/0742-8413(95)00039-q" @default.
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