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- W1992175080 abstract "Helicobacter pylori eradication has a differential effect on the facets of idiopathic parkinsonism (IP): brady/hypokinesia improves, but rigidity worsens. Small intestinal bacterial overgrowth is common in IP and has been described as a sequel to Helicobacter eradication. The hyperhomocysteinaemia of IP is, in part, explained by serum vitamin B(12), but the concentration is not explained by Helicobacter status. Moreover, Helicobacter-associated gastric atrophy is uncommon in IP. However, overgrowth both increases B(12) utilization and provides a source of inflammation to drive homocysteine production. It is not a bystander event in IP: clouds of lysosomes are seen in duodenal enterocytes. Its candidature for causality of a rigidity-associated pathway is circumstantial: there are biological gradients of rigidity on natural killer and T-helper blood counts, both being higher with hydrogen breath test positivity for overgrowth." @default.
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- W1992175080 date "2012-01-01" @default.
- W1992175080 modified "2023-09-26" @default.
- W1992175080 title "Towards Defining a Rigidity-Associated Pathogenic Pathway in Idiopathic Parkinsonism" @default.
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- W1992175080 doi "https://doi.org/10.1159/000332807" @default.
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