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- W1992221035 abstract "Background: Catecholamines and angiotensin II overproduction reduces fluid delivery to the Henle's loop and renal excretion of solute-free water. In ascitic cirrhosis, hypersecretion of vasopressin (ADH) is thought to rule tubular free-water reabsorption (TFWR), but ADH V2-receptor antagonists are not beneficial in the long-term treatment of hyponatremic ascites." @default.
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- W1992221035 date "2014-02-01" @default.
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- W1992221035 title "Pathogenesis of solute-free water retention in experimental ascitic cirrhosis: Is vasopressin (ADH) the only agent to blame?" @default.
- W1992221035 doi "https://doi.org/10.1016/j.dld.2014.01.080" @default.
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