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- W1992810984 abstract "Muscle atrophy is a widespread clinical finding common to many congenital and acquired conditions. Maintaining mobility and muscle integrity is crucial for the management and quality of life of these patients. Previous research indicated that neuronal nitric oxide synthase (nNOS) contributes to preservation of muscle mass and blood flow. In healthy muscle, nNOS is associated with the dystrophin–glycoprotein complex (DGC) at the sarcolemma via interactions with alpha-syntrophin. Loss of sarcolemmal nNOS is associated with atrophy in hindlimb suspension mouse models and exercise-induced decrease in muscle blood flow and fatigue in dystrophin-deficient muscular dystrophies, such as Duchenne muscular dystrophy." @default.
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- W1992810984 date "2010-10-01" @default.
- W1992810984 modified "2023-09-26" @default.
- W1992810984 title "P3.19 Decrease of sarcolemmal nNOS as a molecular marker of muscle atrophy in inherited and acquired forms of myopathy" @default.
- W1992810984 doi "https://doi.org/10.1016/j.nmd.2010.07.161" @default.
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