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- W1993669933 abstract "Apoptotic cell death is a critical feature of the regulated development of multicellular organisms. An individual cell must integrate signals delivered by death-inducing and survival-promoting receptors to determine its fate in the context of the organism as a whole. A major focus on the regulation of apoptosis has been on the identification of new molecules that influence cell survival in either a positive or a negative fashion. One of the best studied survival genes is bcl-2, the first member identified of a growing family of genes that participate in the control of apoptosis. The bcl-2 gene family contains both anti-apoptotic and pro-apoptotic members (for review, seeYang and Korsmeyer 1996). To date, most studies investigating the function of these genes have relied on simple overexpression. However, the levels of expression of Bcl-2-related proteins do not always predict the ability of a cell to resist death-promoting stimuli. Such data have been interpreted as evidence for Bcl-2-independent mechanisms of regulating apoptosis (Strasser et al. 1995)." @default.
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- W1993669933 date "1996-11-01" @default.
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- W1993669933 title "Apoptosis Meets Signal Transduction: Elimination of a BAD Influence" @default.
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- W1993669933 doi "https://doi.org/10.1016/s0092-8674(00)81377-x" @default.
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