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• W1994179035 startingPage "92" @default.
• W1994179035 abstract "The effect of glycine in preventing cadmium (Cd) teratogenicity in mice was studied. Cadmium chloride (CdCl2) was administered subcutaneously at 1, 2 or 4 mg/kg doses on gestation days (GD) 7, 8 and 9. Glycine was given ad libitum (in the drinking water) from GD0 through GD18 (the day when animals were killed), as a 1% and 2% drinking water solution. Cd and nucleic acid concentrations in embryos were determined. The most common finding seen after CdCl2 4 mg/kg exposure was exencephaly. The incidence of this malformation was significantly reduced in mice receiving 2% glycine while fetal Cd significantly decreased as compared to cadmium-treated positive control animals. Increased nucleic acid levels were seen in the same embryos. In glycine non-supplemented mice given CdCl2 4 mg/kg, embryonic lipid peroxidation proved to be increased. In conclusion, lipid peroxidation was associated with cadmium-induced teratogenicity, and glycine inhibited the cadmium-induced effect by inhibiting placental transport of cadmium. However, further detailed studies are needed to establish the mechanism(s) of action." @default.
• W1994179035 created "2016-06-24" @default.
• W1994179035 creator A5011467041 @default.
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• W1994179035 date "2007-01-01" @default.
• W1994179035 modified "2023-10-16" @default.
• W1994179035 title "Glycine reduces cadmium-induced teratogenic damage in mice" @default.
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• W1994179035 doi "https://doi.org/10.1016/j.reprotox.2006.08.011" @default.
• W1994179035 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/17034988" @default.
• W1994179035 hasPublicationYear "2007" @default.
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