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- W1994362420 abstract "The lung is one of the organs most sensitive to irradiation, and the application of radiation to eradicate malignant disease is limited by this sensitivity, so that optimal doses may not be practicable. Pulmonary fibrosis is a potentially lethal and chronic consequence of such anti-tumour therapy. Irradiation is often administered in conjunction with chemotherapy in combined modality therapies, and many different cellular systems which interact in lung tissue may be affected. The ‘late’ clinical manifestations of radiation damage to lung tissue, pneumonitis and fibrosis, have been studied clinically and in the laboratory using a variety of dose/time schedules. The results of this research have led to the proposition that pneumonitis and fibrosis arise from the effects of irradiation on different target cells, Type II pneumocytes and septal fibroblasts, respectively; and that the two reactions occur at different and well-defined points in time. More recent evidence suggests that these processes each involve several cell systems, and sophisticated intercellular communications too. Moreover the two reactions are inter-related. Fibrosis invariably occurs after pneumonitis, but can also occur without the appearance of clinical pneumonitis." @default.
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- W1994362420 date "1994-03-01" @default.
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- W1994362420 title "Normal lung biology" @default.
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- W1994362420 doi "https://doi.org/10.1016/0169-5002(94)91666-7" @default.
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