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• W1994501076 abstract "Excessive superoxide production after cerebral ischemia is known to mediate neuronal injury. Angiotensin II type 1 receptor activation results in production of superoxide, but whether angiotensin II type 1 receptor blockade prevents production of superoxide and subsequent neuronal injury after ischemia remains unclear. Normotensive rats received the angiotensin II type 1 receptor blocker, candesartan or only vehicle before induction of global cerebral ischemia. Approximately 30% of the hippocampal CA1 neurons survived in candesartan-treated animals, whereas only 2% of neurons survived in vehicle-treated animals. Superoxide production was significantly less in these vulnerable neurons in candesartan-treated animals than in vehicle-treated animals. Angiotensin II type 1 receptor may have an essential role in superoxide production and subsequent injury in vulnerable neurons after global cerebral ischemia." @default.
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• W1994501076 date "2005-03-01" @default.
• W1994501076 modified "2023-09-27" @default.
• W1994501076 title "Candesartan reduces superoxide production after global cerebral ischemia" @default.
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• W1994501076 doi "https://doi.org/10.1097/00001756-200503150-00004" @default.
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