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• W1994646972 abstract "CCL3/macrophage inflammatory protein (MIP-1)-1α is elevated in the rectal biopsies of patients with active inflammatory bowel diseases, but its role remains undefined. The present study examined the role of CCL3/MIP-1α during trinitrobenzene sulfonic acid (TNBS)-induced colitis in the rat. Colonic CCL3/MIP-1α levels were elevated (>20-fold above control) within 24 h and remained elevated to day 7 of colitis induction by TNBS administration. In addition, significant increases in colonic neutrophil accumulation were observed within 24 h to day 7 of TNBS treatment. Pre-treatment of rats with a single dose of CCL3/MIP-1α antibody significantly reduced (47%) colonic neutrophil accumulation during the early (24 h) phase of TNBS-induced colitis. In contrast, chronic (repeated) administration of CCL3/MIP-1α antibody did not attenuate colonic neutrophil accumulation during the late phase (day 7) of TNBS-induced colitis. These results suggest a role for CCL3/MIP-1α in promoting colonic neutrophil accumulation during the early (24 h) phase of TNBS-induced colitis." @default.
• W1994646972 created "2016-06-24" @default.
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• W1994646972 date "2004-08-01" @default.
• W1994646972 modified "2023-10-18" @default.
• W1994646972 title "The role of CCL3/macrophage inflammatory protein-1α in experimental colitis" @default.
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• W1994646972 doi "https://doi.org/10.1016/j.ejphar.2004.07.005" @default.
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