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• W1994749069 abstract "ObjectiveTo evaluate maternal hyperoxygenation test (MHT) in predicting the outcome of fetuses at risk for neonatal pulmonary hypoplasia.Study designMHT test was performed on 9 pregnant women (median maternal age 28.5 years) with fetuses with congenital anomalies(median gestation 31.5 weeks) that may cause pulmonary hypoplasia.(hypoplastic left heart syndrome(n = 2), congenital diaphragmatic hernia (left) (n = 2), Epstein anomaly with pulmonary artesia (n = 1), tetralogy of Fallot with absent pulmonary valve (n = 1), prune belly syndrome (n = 1), lung hypoplasia of unknown etiology (n = 2).During MHT the Doppler blood velocity pattern and PI in the first branch of either pulmonary arteries were obtained before and 10-15 minutes after maternal breathing of 100% oxygen was obtained for each fetus at ≥30weeks GA. An increase in fetal blood flow with oxygen (a decrease of ≥20% in PI) was considered positive for pulmonary reactivity. The primary outcome for this study was neonatal death from possible pulmonary hypoplasia.ResultsOf the 9 fetuses tested, 5 (55%) had a reactive MHT and all were delivered and alive and well (100%). Of the 4 fetuses (45%) who had a non reactive MHT 2 died in the early neonatal period (50%) and 1 (25%) was born alive but no data was available for followup, 1 (25%) is alive and well.ConclusionPreliminary data suggest that testing fetal pulmonary vascular reactivity with MHT could be useful in assessing fetal pulmonary vascular resistance prenatally and in predicting the neonatal outcome. More data is needed to assess the limitations of this test. ObjectiveTo evaluate maternal hyperoxygenation test (MHT) in predicting the outcome of fetuses at risk for neonatal pulmonary hypoplasia. To evaluate maternal hyperoxygenation test (MHT) in predicting the outcome of fetuses at risk for neonatal pulmonary hypoplasia. Study designMHT test was performed on 9 pregnant women (median maternal age 28.5 years) with fetuses with congenital anomalies(median gestation 31.5 weeks) that may cause pulmonary hypoplasia.(hypoplastic left heart syndrome(n = 2), congenital diaphragmatic hernia (left) (n = 2), Epstein anomaly with pulmonary artesia (n = 1), tetralogy of Fallot with absent pulmonary valve (n = 1), prune belly syndrome (n = 1), lung hypoplasia of unknown etiology (n = 2).During MHT the Doppler blood velocity pattern and PI in the first branch of either pulmonary arteries were obtained before and 10-15 minutes after maternal breathing of 100% oxygen was obtained for each fetus at ≥30weeks GA. An increase in fetal blood flow with oxygen (a decrease of ≥20% in PI) was considered positive for pulmonary reactivity. The primary outcome for this study was neonatal death from possible pulmonary hypoplasia. MHT test was performed on 9 pregnant women (median maternal age 28.5 years) with fetuses with congenital anomalies(median gestation 31.5 weeks) that may cause pulmonary hypoplasia.(hypoplastic left heart syndrome(n = 2), congenital diaphragmatic hernia (left) (n = 2), Epstein anomaly with pulmonary artesia (n = 1), tetralogy of Fallot with absent pulmonary valve (n = 1), prune belly syndrome (n = 1), lung hypoplasia of unknown etiology (n = 2).During MHT the Doppler blood velocity pattern and PI in the first branch of either pulmonary arteries were obtained before and 10-15 minutes after maternal breathing of 100% oxygen was obtained for each fetus at ≥30weeks GA. An increase in fetal blood flow with oxygen (a decrease of ≥20% in PI) was considered positive for pulmonary reactivity. The primary outcome for this study was neonatal death from possible pulmonary hypoplasia. ResultsOf the 9 fetuses tested, 5 (55%) had a reactive MHT and all were delivered and alive and well (100%). Of the 4 fetuses (45%) who had a non reactive MHT 2 died in the early neonatal period (50%) and 1 (25%) was born alive but no data was available for followup, 1 (25%) is alive and well. Of the 9 fetuses tested, 5 (55%) had a reactive MHT and all were delivered and alive and well (100%). Of the 4 fetuses (45%) who had a non reactive MHT 2 died in the early neonatal period (50%) and 1 (25%) was born alive but no data was available for followup, 1 (25%) is alive and well. ConclusionPreliminary data suggest that testing fetal pulmonary vascular reactivity with MHT could be useful in assessing fetal pulmonary vascular resistance prenatally and in predicting the neonatal outcome. More data is needed to assess the limitations of this test. Preliminary data suggest that testing fetal pulmonary vascular reactivity with MHT could be useful in assessing fetal pulmonary vascular resistance prenatally and in predicting the neonatal outcome. More data is needed to assess the limitations of this test." @default.
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• W1994749069 title "Prenatal prediction of pulmonary hypoplasia using the maternal hyperoxygenation test and neonatal outcome" @default.
• W1994749069 doi "https://doi.org/10.1016/j.ajog.2004.10.430" @default.
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