FRINK Linked Data Fragments server

Matches in SemOpenAlex for { <https://semopenalex.org/work/W1994880957> ?p ?o ?g. }

• W1994880957 endingPage "962" @default.
• W1994880957 startingPage "955" @default.
• W1994880957 abstract "ObjectivePrevious reports from select hypertensive patients suggest that atherosclerotic renovascular disease (RVD) is rapidly progressive and associated with a decline in kidney size and kidney function. This prospective, population-based study estimates the incidence of new RVD and progression of established RVD among elderly, free-living participants in the Cardiovascular Health Study (CHS).MethodThe CHS is a multicenter, longitudinal cohort study of cardiovascular risk factors, morbidity, and mortality among men and women aged >65 years old. From 1995 through 1996, 834 participants underwent renal duplex sonography (RDS) to define the presence or absence of significant RVD. Between 2002 and 2005, a second RDS study was performed in 119 participants (mean study interval, 8.0 ± 0.8 years). Significant RVD was defined as hemodynamically significant stenosis (renal artery peak systolic velocity [RA-PSV] exceeding 1.8 m/s) or renal artery occlusion. Prevalent RVD was significant RVD at the first RDS, and incident disease was defined as new significant RVD at the second RDS. Significant change of RVD was defined as a change in RA-PSV of greater than two times the standard deviation of expected change over time, regardless of hemodynamic significance or progression to renal artery occlusion.ResultsThe second RDS study cohort included 119 CHS participants with 235 kidneys (35% men; mean age, 82.8 ± 3.4). On follow-up, no prevalent RVD (n = 13 kidneys; 6.0%) progressed to occlusion. Twenty-nine kidneys without RVD at the first RDS demonstrated significant change in PSV at the second RDS; including nine kidneys with new significant RVD (8 new stenoses; 1 new occlusion). Controlling for within-subject correlation, the overall estimated change in RVD among all 235 kidneys was 14.0% (95% confidence interval [CI], 9.2% to 21.4%), with progression to significant RVD in 4.0% (95% CI, 1.9% to 8.2%). Longitudinal increase in diastolic blood pressure and decrease in renal length were significantly associated with progression to new (ie, incident) significant RVD but not prevalent RVD.ConclusionsThis is the first prospective, population-based estimate of incident RVD and progression of prevalent RVD among free-living elderly Americans. In contrast to previous reports among select hypertensive patients, CHS participants with a low rate of clinical hypertension demonstrated a significant change of RVD in only 14.0% of kidneys on follow-up of 8 years (annualized rate, 1.3% per year). Progression to significant RVD was observed in only 4.0% (annualized rate, 0.5% per year), and no prevalent RVD progressed to occlusion. Previous reports from select hypertensive patients suggest that atherosclerotic renovascular disease (RVD) is rapidly progressive and associated with a decline in kidney size and kidney function. This prospective, population-based study estimates the incidence of new RVD and progression of established RVD among elderly, free-living participants in the Cardiovascular Health Study (CHS). The CHS is a multicenter, longitudinal cohort study of cardiovascular risk factors, morbidity, and mortality among men and women aged >65 years old. From 1995 through 1996, 834 participants underwent renal duplex sonography (RDS) to define the presence or absence of significant RVD. Between 2002 and 2005, a second RDS study was performed in 119 participants (mean study interval, 8.0 ± 0.8 years). Significant RVD was defined as hemodynamically significant stenosis (renal artery peak systolic velocity [RA-PSV] exceeding 1.8 m/s) or renal artery occlusion. Prevalent RVD was significant RVD at the first RDS, and incident disease was defined as new significant RVD at the second RDS. Significant change of RVD was defined as a change in RA-PSV of greater than two times the standard deviation of expected change over time, regardless of hemodynamic significance or progression to renal artery occlusion. The second RDS study cohort included 119 CHS participants with 235 kidneys (35% men; mean age, 82.8 ± 3.4). On follow-up, no prevalent RVD (n = 13 kidneys; 6.0%) progressed to occlusion. Twenty-nine kidneys without RVD at the first RDS demonstrated significant change in PSV at the second RDS; including nine kidneys with new significant RVD (8 new stenoses; 1 new occlusion). Controlling for within-subject correlation, the overall estimated change in RVD among all 235 kidneys was 14.0% (95% confidence interval [CI], 9.2% to 21.4%), with progression to significant RVD in 4.0% (95% CI, 1.9% to 8.2%). Longitudinal increase in diastolic blood pressure and decrease in renal length were significantly associated with progression to new (ie, incident) significant RVD but not prevalent RVD. This is the first prospective, population-based estimate of incident RVD and progression of prevalent RVD among free-living elderly Americans. In contrast to previous reports among select hypertensive patients, CHS participants with a low rate of clinical hypertension demonstrated a significant change of RVD in only 14.0% of kidneys on follow-up of 8 years (annualized rate, 1.3% per year). Progression to significant RVD was observed in only 4.0% (annualized rate, 0.5% per year), and no prevalent RVD progressed to occlusion." @default.
• W1994880957 created "2016-06-24" @default.
• W1994880957 creator A5008129967 @default.
• W1994880957 creator A5033559569 @default.
• W1994880957 creator A5035154917 @default.
• W1994880957 creator A5040129941 @default.
• W1994880957 creator A5069415556 @default.
• W1994880957 creator A5081362731 @default.
• W1994880957 creator A5085936810 @default.
• W1994880957 date "2006-11-01" @default.
• W1994880957 modified "2023-09-25" @default.
• W1994880957 title "Progression of atherosclerotic renovascular disease: a prospective population-based study" @default.
• W1994880957 cites W1968419780 @default.
• W1994880957 cites W1973178970 @default.
• W1994880957 cites W1978136073 @default.
• W1994880957 cites W1986544508 @default.
• W1994880957 cites W2001890682 @default.
• W1994880957 cites W2002245828 @default.
• W1994880957 cites W2007242346 @default.
• W1994880957 cites W2009993572 @default.
• W1994880957 cites W2023319119 @default.
• W1994880957 cites W2032556546 @default.
• W1994880957 cites W2036990388 @default.
• W1994880957 cites W2037989593 @default.
• W1994880957 cites W2052824929 @default.
• W1994880957 cites W2054890799 @default.
• W1994880957 cites W2059091616 @default.
• W1994880957 cites W2061701832 @default.
• W1994880957 cites W2069714065 @default.
• W1994880957 cites W2070904782 @default.
• W1994880957 cites W2078249135 @default.
• W1994880957 cites W2081219450 @default.
• W1994880957 cites W2082246284 @default.
• W1994880957 cites W2090140552 @default.
• W1994880957 cites W2111053980 @default.
• W1994880957 cites W2120862215 @default.
• W1994880957 cites W2122238500 @default.
• W1994880957 cites W2123258346 @default.
• W1994880957 cites W2130076239 @default.
• W1994880957 cites W2161197885 @default.
• W1994880957 cites W2171929067 @default.
• W1994880957 cites W2316167515 @default.
• W1994880957 cites W2463163352 @default.
• W1994880957 cites W4234221464 @default.
• W1994880957 cites W4235047721 @default.
• W1994880957 doi "https://doi.org/10.1016/j.jvs.2006.07.031" @default.
• W1994880957 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/16982169" @default.
• W1994880957 hasPublicationYear "2006" @default.
• W1994880957 type Work @default.
• W1994880957 sameAs 1994880957 @default.
• W1994880957 citedByCount "53" @default.
• W1994880957 countsByYear W19948809572012 @default.
• W1994880957 countsByYear W19948809572013 @default.
• W1994880957 countsByYear W19948809572014 @default.
• W1994880957 countsByYear W19948809572016 @default.
• W1994880957 countsByYear W19948809572017 @default.
• W1994880957 countsByYear W19948809572019 @default.
• W1994880957 countsByYear W19948809572022 @default.
• W1994880957 crossrefType "journal-article" @default.
• W1994880957 hasAuthorship W1994880957A5008129967 @default.
• W1994880957 hasAuthorship W1994880957A5033559569 @default.
• W1994880957 hasAuthorship W1994880957A5035154917 @default.
• W1994880957 hasAuthorship W1994880957A5040129941 @default.
• W1994880957 hasAuthorship W1994880957A5069415556 @default.
• W1994880957 hasAuthorship W1994880957A5081362731 @default.
• W1994880957 hasAuthorship W1994880957A5085936810 @default.
• W1994880957 hasBestOaLocation W19948809571 @default.
• W1994880957 hasConcept C126322002 @default.
• W1994880957 hasConcept C141071460 @default.
• W1994880957 hasConcept C159641895 @default.
• W1994880957 hasConcept C164705383 @default.
• W1994880957 hasConcept C188816634 @default.
• W1994880957 hasConcept C2776634560 @default.
• W1994880957 hasConcept C2778653478 @default.
• W1994880957 hasConcept C2780091579 @default.
• W1994880957 hasConcept C2781016617 @default.
• W1994880957 hasConcept C2908647359 @default.
• W1994880957 hasConcept C71924100 @default.
• W1994880957 hasConcept C72563966 @default.
• W1994880957 hasConcept C99454951 @default.
• W1994880957 hasConceptScore W1994880957C126322002 @default.
• W1994880957 hasConceptScore W1994880957C141071460 @default.
• W1994880957 hasConceptScore W1994880957C159641895 @default.
• W1994880957 hasConceptScore W1994880957C164705383 @default.
• W1994880957 hasConceptScore W1994880957C188816634 @default.
• W1994880957 hasConceptScore W1994880957C2776634560 @default.
• W1994880957 hasConceptScore W1994880957C2778653478 @default.
• W1994880957 hasConceptScore W1994880957C2780091579 @default.
• W1994880957 hasConceptScore W1994880957C2781016617 @default.
• W1994880957 hasConceptScore W1994880957C2908647359 @default.
• W1994880957 hasConceptScore W1994880957C71924100 @default.
• W1994880957 hasConceptScore W1994880957C72563966 @default.
• W1994880957 hasConceptScore W1994880957C99454951 @default.
• W1994880957 hasIssue "5" @default.
• W1994880957 hasLocation W19948809571 @default.
• W1994880957 hasLocation W19948809572 @default.
• W1994880957 hasOpenAccess W1994880957 @default.
• W1994880957 hasPrimaryLocation W19948809571 @default.

• next