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- W1995173863 abstract "The recently reported observations of Correia and Delgado1add convincing support to other reports2,3that an abnormal T-cell response is involved in the pathogenesis of toxic epidermal necrolysis (TEN). The observations that support the theory of immune system mediation of this disease include the following: The occurrence of TEN as a manifestation of severe acute graft-vs-host disease suggests a cell-mediated immune pathogenesis. The dermal infiltrate in TEN is composed mainly of activated T lymphocytes with a predominantly cytotoxic phenotype. Patients who suffer a second bout of TEN caused by the same drug undergo a shorter incubation period and have more severe disease. Keratinocytes express HLA-DR and ICAM-14molecules; normally, they do not. Recently, in the early lesions of TEN, perforin mRNA has been found in the granules of CD8 cytotoxic T cells.5 In view of these facts, it has been unfortunate that in the" @default.
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- W1995173863 date "1994-01-01" @default.
- W1995173863 modified "2023-09-27" @default.
- W1995173863 title "Abnormal T-Cell Response in Toxic Epidermal Necrolysis" @default.
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- W1995173863 doi "https://doi.org/10.1001/archderm.1994.01690010122026" @default.
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