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• W1995487952 abstract "Acetaminophen (APAP) produces antinociception and hypothermia. Because the antinociceptive effect in rats is partially dependent on opioid and cannabinoid CB1 receptor activation, we determined if activation of these receptors also contributes to the hypothermic effect of APAP. Rats injected with APAP (100, 250, 375 or 500 mg/kg, i.p.) displayed dose-related hypothermia. For combined administration, the hypothermic effect of APAP (400 mg/kg, i.p.) was not altered by pretreatment with: naltrexone (10 mg/kg, s.c.), a non-selective opioid antagonist; naltrindole (1 mg/kg, s.c.), a delta opioid antagonist; nor-binaltorphimine (10 mg/kg, i.p.), a kappa opioid antagonist; SR 141716A (3 mg/kg, i.m.), a cannabinoid CB1 receptor antagonist; or JTC-801(1 mg/kg, i.p.), a nociceptin/orphanin FQ peptide (NOP) receptor antagonist. The demonstration that APAP produces hypothermia independent of opioid, cannabinoid CB1 or NOP receptor activation is contrary to its antinociceptive effect, which requires opioid and cannabinoid CB1 receptor activation." @default.
• W1995487952 created "2016-06-24" @default.
• W1995487952 creator A5039321173 @default.
• W1995487952 creator A5085810341 @default.
• W1995487952 date "2009-05-01" @default.
• W1995487952 modified "2023-09-27" @default.
• W1995487952 title "Opioid, cannabinoid CB1 and NOP receptors do not mediate APAP-induced hypothermia in rats" @default.
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• W1995487952 doi "https://doi.org/10.1016/j.pbb.2009.01.020" @default.
• W1995487952 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/19463266" @default.
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