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• W1995882260 abstract "The CD5+B lymphocyte (B1a) population is known to be involved in most immune responses to microorganism TI antigens. Moreover, xid mice deficient for immune responses against TI-2 antigens are known to lack the B1a population, suggesting a role for B1a cells in TI-2 immune responses. We previously established that the oligosaccharide human blood group A antigen stimulated murine TI-2 immune responses. In this work, we show that the frequency of anti-A-secreting hybridomas was higher in mice with larger splenic B1a populations and that in vivo anti-CD5 treatment reduced anti-A immune response without affecting the response against TD RBC antigens. A similar effect was observed by in vitro anti-CD5 treatment of splenocytes. The in vivo anti-CD5 treatment also interfered with the immunization-dependent increase in splenocyte numbers. These results are in agreement with an important role for the B-cell CD5 receptor in the regulation of TI-2 immune responses possibly mediated by its interaction with the CD72 ligand." @default.
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• W1995882260 date "1997-01-01" @default.
• W1995882260 modified "2023-10-09" @default.
• W1995882260 title "CD5⁺B Cell-Dependent Regulation of the Murine T-Cell Independent Immune Response Against the Human Blood Group a Antigen" @default.
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• W1995882260 doi "https://doi.org/10.3109/08820139709088546" @default.
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