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Matches in SemOpenAlex for { <https://semopenalex.org/work/W1997048360> ?p ?o ?g. }

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• W1997048360 startingPage "439" @default.
• W1997048360 abstract "The 5'-flanking sequences of the human macrophage inflammatory protein-3alpha/CCL20 gene were cloned and transfected into G-361 human melanoma cells in a luciferase reporter construct. Tumor necrosis factor-alpha (TNF-alpha) treatment stimulated luciferase expression, and promoter truncations demonstrated that TNF-alpha inducibility is conferred by a region between nt -111 and -77, which contains a non-standard nuclear factor-kappaB (NF-kappaB) binding site. The requirement for NF-kappaB was demonstrated as follows: (i) mutations in this NF-kappaB site abrogated TNF-alpha responsiveness; (ii) TNF-alpha activated a construct containing two copies of the CCL20 NF-kappaB binding site; (iii) overexpression of NF-kappaB p65 activated the CCL20 promoter; (iv) NF-kappaB from nuclear extracts of TNF-alpha-stimulated cells bound specifically to this NF-kappaB site." @default.
• W1997048360 created "2016-06-24" @default.
• W1997048360 creator A5003539515 @default.
• W1997048360 creator A5065078406 @default.
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• W1997048360 date "2001-11-27" @default.
• W1997048360 modified "2023-10-16" @default.
• W1997048360 title "Human macrophage inflammatory protein-3α/CCL20/LARC/Exodus/SCYA20 is transcriptionally upregulated by tumor necrosis factor-α via a non-standard NF-κB site" @default.
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• W1997048360 doi "https://doi.org/10.1016/s0014-5793(01)03138-6" @default.
• W1997048360 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/11749970" @default.
• W1997048360 hasPublicationYear "2001" @default.
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