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- W1997610080 endingPage "4006" @default.
- W1997610080 startingPage "3989" @default.
- W1997610080 abstract "Bile duct ligation (BDL)-treated rats exhibit cholestasis, increased systemic oxidative stress, and liver fibrosis, which ultimately lead to liver cirrhosis. Asymmetric dimethylarginine (ADMA) is a competitive inhibitor of nitric oxide synthase that can decrease the synthesis of nitric oxide. BDL rats have higher plasma and hepatic ADMA levels, which may be due to increased hepatic protein arginine methyltransferase-1 and decreased dimethylarginine dimethylaminohydrolase expression. BDL rats also exhibit renal and brain damage characterized by increased tissue ADMA concentrations. The increased plasma ADMA levels and multiple organ damages seen here are also observed following multiple organ failures associated with critical illness. This review discusses the dysregulation of ADMA in major organs in BDL rats and the role of increased ADMA in multiple organ damages." @default.
- W1997610080 created "2016-06-24" @default.
- W1997610080 creator A5016188669 @default.
- W1997610080 creator A5018504378 @default.
- W1997610080 creator A5029527384 @default.
- W1997610080 creator A5081155708 @default.
- W1997610080 date "2014-03-05" @default.
- W1997610080 modified "2023-10-14" @default.
- W1997610080 title "Increased Circulatory Asymmetric Dimethylarginine and Multiple Organ Failure: Bile Duct Ligation in Rat as a Model" @default.
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