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• W1997788251 abstract "Glucose-free medium (aglycaemia) caused a complete failure of CA1 population spikes (after 14.5 +/- 0.8 min) and field EPSPs (after 18.1 +/- 0.5 min). In the presence of the selective adenosine A1 antagonist, 8-(p-sulfophenyl)theophylline (10 microM), population spikes and EPSPs were decreased by only 13.8 +/- 11.9% and 32.4 +/- 11.6% at the end of 17.0 +/- 3.0 min and 19.8 +/- 1.7 min of aglycaemia, respectively. A similar effect was produced by caffeine (0.2 mM). The ATP-sensitive K+ channel blockers tolbutamide (1 mM) and glibenclamide (10 microM) had no significant effect on aglycaemic suppression of synaptic transmission. These observations indicate that endogenous adenosine, but not ATP-sensitive K+ conductance, plays a major role in hypoglycaemia failure of transmission." @default.
• W1997788251 created "2016-06-24" @default.
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• W1997788251 date "1993-06-01" @default.
• W1997788251 modified "2023-10-02" @default.
• W1997788251 title "Adenosine release is a major cause of failure of synaptic transmission during hypoglycaemia in rat hippocampal slices" @default.
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• W1997788251 doi "https://doi.org/10.1016/0304-3940(93)90689-i" @default.
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