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- W1998065992 abstract "Monophosphoryl lipid A (MLA), a substructure of bacterial lipopolysaccharide (LPS), is being developed as a prophylactic for sepsis and septic shock. In the present study it was shown that MLA induced a rapid accumulation of IFN-γ in mice that correlated with an in vivo priming of macrophages. Primed macrophages could be induced in vitro to synthesize nitric oxide, a key mediator of macrophage cytotoxicity. Due to its rapid clearance, MLA was not present in circulation at the time when IFN-γ accumulated, suggesting that MLA could not synergize with IFN-γ to systemically activate macrophages in vivo. MLA treatment tolerized mice against the IFN-γ response - ie., treatment of mice with MLA on day 1 blocked LPS from inducing IFN-γ on days 2–4. The significance of these results in relation to MLA's ability to enhance non-specific resistance and block LPS lethality in animals is discussed." @default.
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- W1998065992 title "A rationale for the prophylactic use of monophosphoryl lipid A in sepsis and septic shock" @default.
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- W1998065992 doi "https://doi.org/10.1016/s0006-291x(05)80140-8" @default.
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