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- W1998324306 abstract "Significance Maintaining physiological balance is vital in the primary response to infectious and other stress stimuli to avert damaging inflammation. Delineation of the cell regulatory processes that control inflammatory processes better enable the development of informed strategies to treat associated pathologies. Toward this end, we identify that the promyelocytic leukemia zinc finger (PLZF) transcription factor limits pathogen-induced inflammation. PLZF stabilizes a repressor complex that encompasses histone deacetylase activity, which modifies the state of chromatin. This activity maintains homeostasis by decreasing the scale of induction of select immune response genes. In the absence of PLZF, the chromatin structure is altered, enabling active transcriptional complexes to immediately assemble on gene promoters, resulting in inordinate production of inflammatory cytokines." @default.
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- W1998324306 date "2015-01-20" @default.
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- W1998324306 title "BTB-ZF transcriptional regulator PLZF modifies chromatin to restrain inflammatory signaling programs" @default.
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- W1998324306 doi "https://doi.org/10.1073/pnas.1409728112" @default.
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