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- W1998372130 abstract "Biliary release of glutathione disulfide (GSSG) and glutathione-S-conjugates by the isolated perfused liver under different metabolic conditions has been studied by several investigators [l-3]. Sies et al. [ 1,3] have shown that efflux of GSSG occurs during oxidation of drugs such as hexobarbital and ethylmorphine as well as during hydroperoxide metabolism. In the isolated liver there is also a transport of reduced glutathione (GSH) into the perfusate space [4]. However, it has also been reported that relatively high concentrations of GSH may be excreted in the bile produced in situ ]5a,bl. The present study, performed mainly with isolated perfused rat liver, concerns the release of both reduced and oxidized glutathione in bile after arsenite exposure. It has previously been shown that arsenic is eliminated via this route [6] and compounds such as selenite are known to interact with arsenite in a way that increases their respective elimination in the bile [7,8]. Furthermore, the form of As, e.g., if complexed to small molecular weight compounds, or the mechanism(s) involved in its biliary excretion, have not been described. Similar studies have indicated an involvement of GSH in the excretion of zinc and other metals [9,10] in rat bile in situ." @default.
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- W1998372130 date "1982-08-23" @default.
- W1998372130 modified "2023-10-03" @default.
- W1998372130 title "GSH release in bile as influenced by arsenite" @default.
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- W1998372130 doi "https://doi.org/10.1016/0014-5793(82)80184-1" @default.
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