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- W1998576798 endingPage "391" @default.
- W1998576798 startingPage "379" @default.
- W1998576798 abstract "Type I interferons (IFN-alpha/beta) are produced upon viral and bacterial infections and play essential roles in host defense. However, since IFN-alpha/beta have multiple regulatory functions on innate and adoptive immunity, dysregulation of the IFN-alpha/beta system both in uninfected hosts and during immune responses against infection can result in immunopathologies. In fact, IFN-alpha/beta therapy often accompanies autoimmune-like symptoms. In this regard, we have recently found that mice lacking IFN regulatory factor (IRF)-2, a negative regulator of IFN-alpha/beta signaling, develop spontaneous, CD8(+) T cell-dependent skin inflammation. This unique animal model, together with other animal models, highlights the importance of the mechanism maintaining the homeostasis in the IFN-alpha/beta system even in the absence of infection." @default.
- W1998576798 created "2016-06-24" @default.
- W1998576798 creator A5042128683 @default.
- W1998576798 date "2002-08-01" @default.
- W1998576798 modified "2023-09-26" @default.
- W1998576798 title "Type I interferons and autoimmunity: lessons from the clinic and from IRF-2-deficient mice" @default.
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