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- W1999700055 endingPage "790" @default.
- W1999700055 startingPage "769" @default.
- W1999700055 abstract "Uncontrolled production of collagen I is the main feature of liver fibrosis. Following a fibrogenic stimulus such as alcohol, hepatic stellate cells (HSC) transform into an activated collagen-producing cell. In alcoholic liver disease, numerous changes in gene expression are associated with HSC activation, including the induction of several intracellular signaling cascades, which help maintain the activated phenotype and control the fibrogenic and proliferative state of the cell. Detailed analyses for understanding the molecular basis of the collagen I gene regulation have revealed a complex process involving reactive oxygen species (ROS) as key mediators. Less is known, however, about the contribution of reactive nitrogen species (RNS). In addition, a series of cytokines, growth factors, and chemokines, which activate extracellular matrix (ECM)-producing cells through paracrine and autocrine loops, contribute to the fibrogenic response." @default.
- W1999700055 created "2016-06-24" @default.
- W1999700055 creator A5010469348 @default.
- W1999700055 creator A5031793251 @default.
- W1999700055 creator A5055941563 @default.
- W1999700055 date "2008-11-01" @default.
- W1999700055 modified "2023-10-16" @default.
- W1999700055 title "Oxidative and Nitrosative Stress and Fibrogenic Response" @default.
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